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Diabetes mellitus type I and II

You probably know that there are two types of diabetes.

At first type I, where the insuline producing cells of the pancreas are damaged or destroyed. This might happen due to an infection with viruses or bacteria, but also in the sense of an autimmune process.

Secondly the type II, where the sufficiency of the insuline decreases. Its duty is, to transport the sugar, solved in the blood, into the cells. Therefore sugar- and insuline molecules have to dock at a cell wall, while the insuline works like a switch at the receptor and so channels the sugar molecule into the inner of the cell.

Both forms of disease , type I and II of the diabetes mellitus, have basically one thing in common: a damage at the 7th thoracal vertebra. I am sorry, not being able to answer one question: why one person, having a damage at the 7th thoracal vertebra, gets a diabetes type I and another a diabetes type II.

The classical medicine believes that the type II is released due to overweight. It further postulates that the insuline receptors lose, due to the nutritional disturbance, their functionality at the cell wall, especially at the muscle cells. A for the diabetes type II typical finding is the hyperinsulinemia (too much insuline in the blood), which means that the insuline level rises unnaturally high. The background for that is that the organism tries to compensate the loss of efficiency by raising the amount of hormones.

One thing is sure out of the sight of the SMT®: there exists no diabetes II without a blockade of the 7th thoracal vertebra (Th7 SMT®).

On the assumption that a disturbance of the receptor exists (like classical medicine presumes), the quality of the insuline receptors at the cell walls should also be steered by the 7th thoracal vertebra segment (a segment is the section of the spinal marrow belonging to the right and left emerging spinal nerves), like it happens at the insuline production in the islands of Langerhans of the pancreas. This could be possible, but I could not say if it is really so.

Here I would give way to another theory from the view of the SMT®. Could this inefficiency of the insuline at the receptor not originate in deviations of the spatial arrangement of the amino acid chains of the insuline? This is why the hormone not fits into the receptor, which is equate with the loss of functionality and efficiency of the hormone. As the production of the insuline takes place in the islands of Langerhans in the pancreas, a blockade of the 7th thoracal-, the pancreas vertebra, could be disadvantegeous on the insuline production in this form, that the spatial arrangement of the molecule structure shows minimal deviations.

In spite of all speculations the following statement is a fact: a type II diabetes can be healed by means of the SMT®. Certainly the chance of healing is much higher at the beginning of the disease than after years or even decades of development, simply because in these cases the responsible damages at joints and spine also are older and with that more complicated to remove.

Also a so-called disposition (tendency - in the patient´s family a diabetes type II happens more often) is not an inevitable fate for the concerned, also to get sick with a diabetes type II. The genetical disposition not takes effect as long as the joints and the spine and with it also the 7th thoracal vertebra remain in order.

Totally different is it with the diabetes type I. Here a complete destruction of the insuline producing cells, the islands of Langerhans, happens, so that a lack of insuline arises. There are two reasons for the destruction of the islands of Langerhans:

In the case of an autoimmune background for a diabetes type I, blockades of vertebrae can lead to inflammations in organs, which damage them more or less. These inflammations lead to a cell decomposition, so that suddenly cell material from the inner of the cell appears in the blood. When this material cannot be decomposed quickly enough, it reacts with the immune system. This reaction also has an inflammatory character, which causes still more cell decomposition, more cell material appears in the blood and so the autoimmune process escalates. Therefore damages at joints and spine have a general starter function for most of the autoimmune processes, also at the diabetes mellitus type I.

If the islands of Langerhans are destroyed, they are finally and forever functionless and do not produce insuline any more, i.e., a type I diabetes is not curable.

A big and very frequent problem at the diabetes type I are heavy and apparently completely unmotivated deviations of the blood sugar, which cannot be explained by the classical medicine, although all rules and measures are considered by the patient. These deviations can be excellently balanced by means of the SMT®, also the need of insuline of the patient decreases obviously. However, a final healing is not possible any more.

How can it now be explained, that unmotivated variations of the blood sugar, from extremely high to extremely low, can be bettered by means of the SMT®? After my reflections it only can be that there still exist small rest functions of single, not yet destroyed insuline producing cells. The question, whether still a rest function exists, which allows the development of small amounts of insuline (which leads to an obviously unexplicable lowering of the blood glucose level), or whether there is absolutely no more rest production left (so that the glucose rises very much), depends on how much the 7th thoracal vertebra, the pancreas vertebra, is blocked. Is the extent of the blockade less, a rest production of insuline can happen (the blood sugar sinks), is the blockade in the contrary heavier, no more rest production is possible any more (the blood sugar rises more).

At all diabetics with diabetes I either an infection or an accident was preceding the diabetes. Let me shortly describe two cases: